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Environment
Pesticides
Chemistry
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Pesticides have long been associated with hormone disrupting (endocrine) properties that promote various molecular changes and disease development.
This study highlights how specific estrogen-like pesticides increase disease risk, particularly hormone-related cancers in women (i.e., breast, ovarian, endometrial) and men (i.e., testicular, prostate).
Like pesticides, endocrine disruptors are xenobiotic (i.e., chemical substances that are foreign to an organism or ecosystem). Many reports show that exposure to endocrine disrupting chemicals can affect human, animal, and environmental health by altering the natural body hormones responsible for conventional reproductive, physical, and mental development.
Endocrine disorders can lead to multiple health problems, including hormone-related cancer development (i.e., thyroid, breast, ovarian, prostate, testicular), reproductive disorders, and diabetes/obesity, which can span generations.
Pesticides are one of the most potent xenoestrogenic compounds because their estrogenic potency and environmental half-life exceed those of other xenoestrogenic compounds. This study focuses on organochlorine pesticides (OCs) and evaluates the chemical effects on the physiological (anatomical) system to increase cancer risk.
Many organochlorine pesticides (OCs) can exist in the body for at least three to six years, in soil for decades, and in water for at least a century. In addition, consumption of food and water resources contaminated with OCs can cause these chemicals to accumulate in the body, leading to biomagnification of OCs.
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